Dysrhythmias Cheat Sheet Free Download
Dysrhythmia or arrhythmia are disturbances in the normal cardiac rhythm of the heart which occurs as a result of alterations within the conduction of electrical impulses. These impulses stimulate and coordinate atrial and ventricular myocardial contractions that provide cardiac output.
We are proud to present a printable cheat sheet which you can use as a guide to help you understand dysrhytmias better. There is also a download link below to be used for PDFs:
Click here: Download Dysrhythmias Cheat Sheet.docx
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Dysrhtyhmia |
Description |
Etiology |
P Wave |
P-R Interval |
QRS |
| Sinus Bradycardia | Slow discharge from SA node. R<60bpm, regular rhythm, may cause decreased CO/hypertension | Sleep, hypothermia hypothyroidism, vagal stimulation, suctioning, increased ICP | Normal | Normal | Normal |
| Sinus Tachycardia | Rapid discharges from SA node.
More than 100bpm, regular rhythm, may cause decreased CO, MI |
Hypotension, hypovolemia, fever, anemia, hypoxia, heart failure | Normal | Normal | Normal |
| Premature Atrial Contraction (PAC) | From an ectopic atrial foci, usually with normal conduction.
Irregular rhythm, impulse may be delayed or nonconducted, varies in rate |
May prelude supraventricular tachycardia. Stimulants, hyperthyroidism, COPD, infection and heart diseases | Abnormal | Variable | Normal |
| Paroxysmal Supraventricular Tachycardia (SVT) | From an ecotopic focus above the bundle of His, “re-entry” rate from 100 to 300/minute, regular rhythm. May decrease CO | Exertion, Emotion, Stimulants, Rheumatic Heart Diseases | Abnormal or Hidden | Variable | Normal |
| Atrial Flutter | Ectopic atrial focus “re-entry” Atrial rate is 250 to 400bpm, usually with slow ventricular response | CAD, Valve Problem, Hyperthyroidism | Saw-tooth Shaped | Variable | Normal |
| Atrial Fibrillation | Total disorganization, atrial electrical activity without effective atrial contraction.
Atrial R 300 to 600/minute. |
Usually heart diseases, also hyperthyroid, infection | Chaotic | Can’t be measured | Normal |
| First Degree AV Block | AB Conduction time is gradually prolonged until an atrial impulse is nonconducted and QRS is dropped then repeats | CAD, drugs, RH | Normal | Greater than 0.20 seconds | Normal |
| Second-Degree AV Block—Type 1 | AV conduction time is gradually prolonged until an atrial impulse is nonconducted and QRS is dropped then repeats | MI, drugs | Normal | Progressive Lengthening | Normal width one not conducted |
| Second Degree AV Block—Type 2 | Atrial impulses dropped, without antecedent lengthening P-R
Certain impulses are not conducted |
CAD, MI, digoxin | Occurs in multiples | Normal or Prolonged | Widened preceded by two or more P waves |
| Third Degree—Complete AV Block | No atrial impulses conducted, atrium and ventricle contract separately, result is decreased CO and heart failure | Calcification of conduction system, CAD, cardiomyopathy | Normal | Variable | Normal or Widened |
| Premature Ventricular Contractions (PVC) | From signle or multiple ectopic focus in ventricle.
Premature and distorted QRS. Rate is 60 to 100bpm and irregular |
Ischemia, Stimulants, hypokalemia, stress and fever | None | Not measurable | Widened and distorted |
| Ventricular Tachycardia | Run of three or more PVCs, ventricular focus or foci fire repeatedly.
Rate is 110 to 250bpm |
MI, CAD, Some drugs | Usually none | Not measurable | Wide and distorted |
| Ventricular Fibrillation | Severe derangement, firing multiple ventricular foci. No effective ventricular contraction.
Terminal if untreated. |
Ischemia, infarction, CAD, cardiomyopathy | None | Not measurable | Wide and Distorted |
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