12 Spinal Cord Injury Nursing Care Plans

May 10, 2014

A spinal cord injury (SCI) is damage to any part of the spinal cord or nerves at the end of the spinal canal. The condition often causes permanent changes in strength, sensation, and other body functions below the site of the injury.

Motor vehicle accidents, acts of violence, and sporting injuries are the common causes of spinal cord injury (SCI). The mechanism of injury influences the type of SCI and the degree of neurological deficit. Spinal cord lesions are classified as a complete (total loss of sensation and voluntary motor function) or incomplete (mixed loss of sensation and voluntary motor function).

Physical findings vary, depending on the level of injury, degree of spinal shock, and phase and degree of recovery, but in general, are classified as follows:

C-1 to C-3: Tetraplegia with total loss of muscular/respiratory function.
C-4 to C-5: Tetraplegia with impairment, reduced pulmonary capacity, complete dependency for ADLs.
C-6 to C-7: Tetraplegia with some arm/hand movement allowing some independence in ADLs.
C-7 to T-1: Tetraplegia with limited use of thumb/fingers, increasing independence.
T-2 to L-1: Paraplegia with intact arm function and varying function of intercostal and abdominal muscles.
L-1 to L-2 or below: Mixed motor-sensory loss; bowel and bladder dysfunction.

Nursing Care Plans

Nursing care planning and goals for patients with spinal cord injuries includes: maximizing respiratory function, preventing injury to the spinal cord, promote mobility and/or independence, prevent or minimize complications, support psychological adjustment of patient and/or SO, and providing information about the injury, prognosis, and treatment.

Here are 12 nursing care plans for patients with spinal cord injury:

Risk for Ineffective Breathing Pattern

Risk for Ineffective Breathing Pattern: The state in which an individual is at high risk to experience an actual or potential loss of adequate ventilation.

Risk factors may include

Impairment of innervation of diaphragm (lesions at or above C-5)
Complete or mixed loss of intercostal muscle function
Reflex abdominal spasms; gastric distension

Possibly evidenced by

[Not applicable; presence of signs and symptoms establishes an actual diagnosis.]

Desired Outcomes

Maintain adequate ventilation as evidenced by absence of respiratory distress and ABGs within acceptable limits
Demonstrate appropriate behaviors to support the respiratory effort.

Nursing Interventions	Rationale
Assess respiratory function by asking patient to take a deep breath. Note presence or absence of spontaneous effort and quality of respirations (labored, using accessory muscles).	C-1 to C-3 injuries result in complete loss of respiratory function. Injuries at C-4 or C-5 can lead to variable loss of respiratory function, depending on phrenic nerve involvement and diaphragmatic function, but generally cause decreased vital capacity and inspiratory effort. For injuries below C-6 or C-7, respiratory muscle function is preserved; however, weakness or impairment of intercostal muscles may impair effectiveness of cough and the ability to sigh, deep breathe.
Auscultate breath sounds. Note areas of absent or decreased breath sounds or development of adventitious sounds (rhonchi).	Hypoventilation is common and leads to accumulation of secretions, atelectasis, and pneumonia (frequent complications). Note: Respiratory compromise is one of the leading causes of mortality, especially during the acute stage as well as later in life.
Note strength or effectiveness of cough.	Level of injury determines the function of intercostal muscles and ability to cough spontaneously or move secretions.
Observe skin color for developing cyanosis, duskiness.	May reveal impending respiratory failure, need for immediate medical evaluation and intervention.
Assess for abdominal distension and muscle spasm.	Abdominal fullness may impede diaphragmatic excursion, reducing lung expansion and further compromising respiratory function.
Monitor and limit visitors as indicated.	General debilitation and respiratory compromise place patient at increased risk for acquiring URIs.
Monitor diaphragmatic movement when phrenic pacemaker is implanted.	Stimulation of phrenic nerve may enhance respiratory effort, decreasing dependency on mechanical ventilator.
Elicit concerns and questions regarding mechanical ventilation devices.	Acknowledges reality of situation.
Provide honest answers.	Future respiratory function needs will not be totally known until spinal shock resolves and acute rehabilitative phase is completed. Even though respiratory support may be required, alternative devices and techniques may be used to enhance mobility and promote independence.
Maintain patent airway: keep head in neutral position, elevate head of bed slightly if tolerated, use airway adjuncts as indicated.	Patients with high cervical injury and impaired gag and cough reflexes require assistance in preventing aspiration and maintaining patient airway.
Assist patient in “taking control” of respirations as indicated. Instruct in and encourage deep breathing, focusing attention on steps of breathing.	Breathing may no longer be a totally voluntary activity but require conscious effort, depending on level of injury and involvement of respiratory muscles.
Assist with coughing as indicated for level of injury (have patient take deep breath and hold for 2 sec before coughing, or inhale deeply, then cough at the end of a slow exhalation). Alternatively, assist by placing hands below diaphragm and pushing upward as patient exhales (quad cough).	Adds volume to cough and facilitates expectoration of secretions or helps move them high enough to be suctioned out. Note: Quad cough procedure is generally reserved for patients with stable injuries once they are in the rehabilitation stage.
Suction as necessary. Document quality and quantity of secretions.	If cough is ineffective, suctioning may be needed to remove secretions, enhance gas exchange, and reduce risk of respiratory infections. Note: “Routine” suctioning increases risk of hypoxia, bradycardia (vagal response), tissue trauma. Therefore, suctioning needs are based on inability to move secretions.
Reposition and turn periodically. Avoid and limit prone position when indicated.	Enhances ventilation of all lung segments, mobilizes secretions, reducing risk of complications such as atelectasis and pneumonia. Note: Prone position significantly decreases vital capacity, increasing risk of respiratory compromise and failure.
Encourage fluids (at least 2000 mL per day).	Aids in liquefying secretions, promoting mobilization and expectoration.
Measure or graph:
Vital capacity (VC), tidal volume (V_T), inspiratory force;	Determines level of respiratory muscle function. Serial measurements may be done to predict impending respiratory failure (acute injury) or determine level of function after spinal shock phase and while weaning from ventilatory support.
Serial ABGs and pulse oximetry.	Documents status of ventilation and oxygenation, identifies respiratory problems such as hypoventilation (low Pao₂and elevated Paco₂) and pulmonary complications.
Administer oxygen by appropriate method (nasal prongs, mask, intubation, ventilator).	Method is determined by level of injury, degree of respiratory insufficiency, and amount of recovery of respiratory muscle function after spinal shock phase.
Assist with use of respiratory adjuncts (incentive spirometer, blow bottles) and aggressive chest physiotherapy (chest percussion).	Preventing retained secretions is essential to maximize gas diffusion and to reduce risk of pneumonia.
Refer and consult with respiratory and physical therapists.	Helpful in identifying exercises individually appropriate to stimulate and strengthen respiratory muscles and effort. For example, glossopharyngeal breathing uses muscles of mouth, pharynx, and larynx to swallow air into lungs, thereby enhancing VC and chest expansion.